An Outline of Psychiatry in Clinical Lectures: The Lectures by Robert Miller ONZM B.A. B.Sc. Ph.D., John Dennison J.P.

By Robert Miller ONZM B.A. B.Sc. Ph.D., John Dennison J.P. M.Sc. B.A.

This paintings is a suite of Carl Wenicke’s lectures on neuropsychiatry translated into English for the 1st time. starting with simple options approximately basic mind functionality, the publication strikes to medical issues, dealing first with continual psychological issues and 'paranoid states', after which to the extra advanced sector of acute psychological issues. the various featured themes are nonetheless clinically suitable, and concerns of latest debate. Carl Wernicke is likely one of the pioneers of neurology and psychiatry; clinicians, researchers and historians will locate this of serious interest.

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Example text

In my experience at least, the characteristic referral to a preferred position in space is totally different from that of remembered visual images. The latter seem to be spatially referred only insofar as they always occupy a definite position in a particular person. They also appear to be of unlimited duration, a difference that is especially difficult to explain. You would hardly imagine that the same perceptual cell, once recruited, would maintain its longlasting state of activation, but rather that it could soon be sensitized by a totally different stimulus.

Even though a few difficulties are raised by these facts, nevertheless the vast number of cell groups needed to accommodate just visual memory images may still seem astonishing. Experience teaches us that a healthy brain can always acquire new memory images; there must therefore still be an abundance of elements for this purpose. However, I draw your attention to a miscalculation that makes us exaggerate the difficulty: As I demonstrate later, we tend to overestimate the number and variability of solid objects in the outside world; and we correspondingly underestimate the frequency of recurrence of the same impressions.

On the one hand, cases have been seen where circumscribed cortical damage in the middle third of the two central gyri leads to permanent deficit, where the hand fails to recognize objects by touch, although disturbance of sensation itself can barely be detected. On the other hand, there are cases of spinal or peripheral disease where sensitivity, and—I stress—also muscle and position sense, is most severely disrupted, with only minor disturbance of tactile perception. The last-named cases demonstrate to some extent that even a very weak and patchy projection system can still conduct messages to the brain sufficient to support primary identification, provided the central projection fields and therefore the remembered images—tactile images, as we can call them here—are preserved.

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